Zulli, Anthony - Victoria University - College of Health & Biomedicine

研究领域

His research focuses on the mechanisms that cardiovascular disease risk factors (homocysteine/cholesterol) cause endothelial dysfunction. His research has an emphasis on the nitric oxide pathway and renin-angiotensin system, and has a recent focus on endothelial derived microparticles. Dr Zulli has been funded by the National Heart Foundation and NHMRC, as well as internal VU grant schemes.

近期论文

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Kruzliak, P., Hare, D.L., Zvonicek, V., Klimas, J., Zulli, A. (2015). Simvastatin impairs the induction of pulmonary fibrosis caused by a western style diet: A preliminary study. J Cell Mol Med. 19(11):2647-54. Alsaadon, H., Kruzliak, P., Smardencas, A., Hayes, A., Bader, M., Angus, P., Herath, C., Zulli, A. (2015). Increased aortic intimal proliferation due to MasR deletion in vitro. Int J Exp Pathol. 96(3):183-7. Berezin, A., Zulli, A., Kerrigan, S., Petrovic, D., Kruzliak, P. (2015). Predictive role of circulating endothelial-derived microparticles in cardiovascular diseases. Clin Biochem. 48(9):562-8. Smith, R.M., Kruzliak, P., Adamcikova, Z., Zulli, A. (2015). Role of Nox inhibitors plumbagin, ML090 and gp91ds-tat peptide on homocysteine thiolactone induced blood vessel dysfunction. Clin Exp Pharmacol Physiol. 42(8):860-4. Habiyakare, B., Alsaadon, H., Mathai, M.L., Hayes, A., Zulli, A. (2014). Reduction of angiotensin A and alamandine vasoactivity in the rabbit model of atherogenesis: differential effects of alamandine and Ang(1-7). Int J Exp Pathol. 95(4):290-5. Wookey, P.J., Zulli, A., Lo, C., Hare, D.L., Schwarer, A.P., Darby, I.A., Leung, A.Y. (2008). Calcitonin receptor (CTR) expression in embryonic, foetal and adult tissues: developmental and pathophysiological implications. In The calcitonin gene-related family: form, function and future perspectives, Eds Drs. Debbie L Hay and Ian M Dickerson. Springer.