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Retraction Note to: IL‑2 augments the sorafenib‑induced apoptosis in liver cancer by promoting mitochondrial fission and activating the JNK/TAZ pathway
Cancer Cell International volume 19, Article number: 358 (2019)
Retraction to: Cancer Cell Int (2018) 18:176 https://doi.org/10.1186/s12935-018-0671-3
The editor has retracted this article [1] because Figures 2 and 4 contain duplicated and modified figures from [2,3,4]. The data reported in this article are therefore unreliable.
Figure 2m in [1] contains figures that have been duplicated and modified from Fig. 2a in [2].
Figure 2 m in [1] contains figures that have been duplicated and modified from Fig. 6j in [3].
Figure 4i in [1] contains figures that are identical to Fig. 4h in [4].
None of the authors have responded to any correspondence from the editor/publisher about this retraction.
References
Ding X, Sun W, Chen J. IL-2 augments the sorafenib-induced apoptosis in liver cancer by promoting mitochondrial fission and activating the JNK/TAZ pathway. Cancer Cell Int. 2018;18:176. https://doi.org/10.1186/s12935-018-0671-3.
Wei R, Cao J, Yao S. Matrine promotes liver cancer cell apoptosis by inhibiting mitophagy and PINK1/Parkin pathways. Cell Stress Chaperones. 2018;23:1295. https://doi.org/10.1007/s12192-018-0937-7.
Jieensinue S, Zhu H, Li G, et al. Tanshinone IIA reduces SW837 colorectal cancer cell viability via the promotion of mitochondrial fission by activating JNK-Mff signaling pathways. BMC Cell Biol. 2018;19:21. https://doi.org/10.1186/s12860-018-0174-z.
Zhang L, Li S, Wang R, Chen C, Ma W, Cai H. Cytokine augments the sorafenib-induced apoptosis in Huh7 liver cancer cell by inducing mitochondrial fragmentation and activating MAPK-JNK signalling pathway. Biomed Pharmacother. 2019;110:213–23. https://doi.org/10.1016/j.biopha.2018.11.037.
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Ding, X., Sun, W. & Chen, J. Retraction Note to: IL‑2 augments the sorafenib‑induced apoptosis in liver cancer by promoting mitochondrial fission and activating the JNK/TAZ pathway. Cancer Cell Int 19, 358 (2019). https://doi.org/10.1186/s12935-019-1083-8
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DOI: https://doi.org/10.1186/s12935-019-1083-8