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Alcohol-induced inhibition of bone formation and neovascularization contributes to the failure of fracture healing via the miR-19a-3p/FOXF2 axis.
Bone & Joint Research ( IF 4.6 ) Pub Date : 2022-6-23 , DOI: 10.1302/2046-3758.116.bjr-2021-0596.r1 Daoyu Zhu 1 , Haoyu Fang 1 , Hongping Yu 2 , Pei Liu 1 , Qianhao Yang 1 , Pengbo Luo 1 , Changqing Zhang 1, 3 , Youshui Gao 1 , Yi-Xuan Chen 1
Bone & Joint Research ( IF 4.6 ) Pub Date : 2022-6-23 , DOI: 10.1302/2046-3758.116.bjr-2021-0596.r1 Daoyu Zhu 1 , Haoyu Fang 1 , Hongping Yu 2 , Pei Liu 1 , Qianhao Yang 1 , Pengbo Luo 1 , Changqing Zhang 1, 3 , Youshui Gao 1 , Yi-Xuan Chen 1
Affiliation
Alcoholism is a well-known detrimental factor in fracture healing. However, the underlying mechanism of alcohol-inhibited fracture healing remains poorly understood.
中文翻译：
酒精诱导的骨形成和新血管形成抑制通过 miR-19a-3p/FOXF2 轴导致骨折愈合失败。
酗酒是众所周知的骨折愈合的有害因素。然而，酒精抑制骨折愈合的潜在机制仍然知之甚少。
更新日期：2022-06-23
中文翻译：
酒精诱导的骨形成和新血管形成抑制通过 miR-19a-3p/FOXF2 轴导致骨折愈合失败。
酗酒是众所周知的骨折愈合的有害因素。然而，酒精抑制骨折愈合的潜在机制仍然知之甚少。
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