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Correction: A novel tumor suppressor protein encoded by circular AKT3 RNA inhibits glioblastoma tumorigenicity by competing with active phosphoinositide-dependent Kinase-1
Molecular Cancer ( IF 37.3 ) Pub Date : 2022-06-07 , DOI: 10.1186/s12943-022-01584-y
Xin Xia 1, 2 , Xixi Li 1, 2 , Fanying Li 1, 2 , Xujia Wu 1, 2 , Maolei Zhang 1, 2 , Huangkai Zhou 1, 2 , Nunu Huang 1, 2 , Xuesong Yang 1, 2 , Feizhe Xiao 3 , Dawei Liu 4 , Lixuan Yang 1, 2 , Nu Zhang 1, 2
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Correction to: Mol Cancer 18, 131 (2019)

https://doi.org/10.1186/s12943-019-1056-5

Following publication of the original article [1], errors were identified in the images presented in Figs. 3 and 4; specifically:

  • Fig. 3C Representative images showing plate colony formations from different cell lines were incorrectly selected during figure assembly; this affects circ-AKT3 IRES mut U373 (bottom left in left-hand colony formations), and circ-AKT3 shRNA control SW1783 and HS683 (top and bottom left in right-hand colony formations). The correct representative images are now used

  • Fig. 4G Representative images of the control group for Empty Vector U373 (bottom left cell line) was incorrectly used. The correct representative image is now used

The corrected figures are provided here. The correction does not have any effect on the results or conclusions of the paper. The original article has been corrected.

  1. Xia X, Li X, Li F, et al. A novel tumor suppressor protein encoded by circular AKT3 RNA inhibits glioblastoma tumorigenicity by competing with active phosphoinositide-dependent Kinase-1. Mol Cancer. 2019;18:131. https://doi.org/10.1186/s12943-019-1056-5.

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Authors and Affiliations

  1. Department of Neurosurgery, The First Affiliated Hospital of Sun Yat-sen University, No 58, Zhongshan 2 Road, Guangzhou, Guangdong Province, 510080, People’s Republic of China

    Xin Xia, Xixi Li, Fanying Li, Xujia Wu, Maolei Zhang, Huangkai Zhou, Nunu Huang, Xuesong Yang & Lixuan Yang

  2. Guangdong Provincial Key Laboratory of Brain Function and Disease, Precise Medicine Institute, The First Affiliated Hospital of Sun Yat-sen University, Guangzhou, Guangdong, 510080, People’s Republic of China

    Xin Xia, Xixi Li, Fanying Li, Xujia Wu, Maolei Zhang, Huangkai Zhou, Nunu Huang, Xuesong Yang & Lixuan Yang

  3. Department of Scientific Research Section, The First Affiliated Hospital of Sun Yat-sen University, Guangzhou, Guangdong Province, 510080, People’s Republic of China

    Feizhe Xiao

  4. Department of Pathology, The First Affiliated Hospital of Sun Yat-sen University, Guangzhou, Guangdong Province, 510080, People’s Republic of China

    Dawei Liu

  5. Department of Neurosurgery, The First Affiliated Hospital of Sun Yat-sen University, No 58, Zhongshan 2 Road, Guangzhou, Guangdong Province, 510080, People’s Republic of China

    Nu Zhang

  6. Guangdong Provincial Key Laboratory of Brain Function and Disease, Precise Medicine Institute, The First Affiliated Hospital of Sun Yat-sen University, Guangzhou, Guangdong, 510080, People’s Republic of China

    Nu Zhang

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Corresponding author

Correspondence to Nu Zhang.

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Xia, X., Li, X., Li, F. et al. Correction: A novel tumor suppressor protein encoded by circular AKT3 RNA inhibits glioblastoma tumorigenicity by competing with active phosphoinositide-dependent Kinase-1. Mol Cancer 21, 124 (2022). https://doi.org/10.1186/s12943-022-01584-y

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中文翻译:

更正:由环状 AKT3 RNA 编码的新型肿瘤抑制蛋白通过与活性磷酸肌醇依赖性激酶-1 竞争抑制胶质母细胞瘤的致瘤性

更正为:Mol Cancer 18, 131 (2019)

https://doi.org/10.1186/s12943-019-1056-5

在原始文章 [1] 发表后,在图 1 和 2 中显示的图像中发现了错误。3和4; 具体来说:

  • 图 3C 显示在图形组装过程中错误选择了来自不同细胞系的平板集落形成的代表性图像;这会影响 circ-AKT3 IRES mut U373(左侧菌落形成中的左下角)和 circ-AKT3 shRNA 对照 SW1783 和 HS683(右侧菌落形成中的左上角和左下角)。现在使用正确的代表性图像

  • 图 4G Empty Vector U373(左下细胞系)对照组的代表性图像使用不正确。现在使用正确的代表图像

此处提供了更正的数字。更正对论文的结果或结论没有任何影响。原文章已更正。

  1. 夏 X,李 X,李 F,等。一种由环状 AKT3 RNA 编码的新型肿瘤抑制蛋白通过与活性磷酸肌醇依赖性激酶 1 竞争来抑制胶质母细胞瘤的致瘤性。摩尔癌症。2019;18:131。https://doi.org/10.1186/s12943-019-1056-5。

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作者和附属机构

  1. 广东省广州市中山二路58号中山大学第一附属医院神经外科 510080

    夏新、李希希、李凡英、吴旭嘉、张茂蕾、周黄凯、黄努努、杨雪松、杨立轩

  2. 中山大学第一附属医院精准医学研究所,广东省脑功能与疾病重点实验室,广东广州,510080

    夏新、李希希、李凡英、吴旭嘉、张茂蕾、周黄凯、黄努努、杨雪松、杨立轩

  3. 中山大学第一附属医院科研科, 广东省广州市, 510080

    肖飞哲

  4. 中山大学第一附属医院病理科,广东广州,510080

    刘大伟

  5. 广东省广州市中山二路58号中山大学第一附属医院神经外科 510080

    张怒

  6. 中山大学第一附属医院精准医学研究所,广东省脑功能与疾病重点实验室,广东广州,510080

    张怒

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夏,X.,李,X.,李,F.等。更正:一种由环状 AKT3 RNA 编码的新型肿瘤抑制蛋白通过与活性磷酸肌醇依赖性激酶 1 竞争来抑制胶质母细胞瘤的致瘤性。摩尔癌症 21, 124 (2022)。https://doi.org/10.1186/s12943-022-01584-y

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更新日期:2022-06-07
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