Ulcerative colitis (UC) is a severe disease of the intestinal tract. To investigate the role of TLR4/Mitogen-activated protein kinase (MAPK)/Nuclear factor kappa-B(NF-κB) pathways and intestinal flora in UC, and the protective mechanisms of Mesona chinensis Benth polysaccharides (MBP), potential therapeutic agents due to their diabetes-relieving, cancer-suppressing, and immunomodulatory properties.

中文翻译：

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Mesona chinensis Benth 多糖通过抑制 TLR4/MAPK/NF-κB 信号通路和调节肠道微生物群来缓解 DSS 诱导的溃疡性结肠炎

溃疡性结肠炎（UC）是一种严重的肠道疾病。探讨 TLR4/丝裂原活化蛋白激酶 (MAPK)/核因子 kappa-B(NF-κB) 通路和肠道菌群在 UC 中的作用，以及中华白芍多糖 (MBP) 的保护机制，这些潜在的治疗药物它们具有缓解糖尿病、抑制癌症和免疫调节的特性。