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LncRNA CAIF suppresses LPS-induced inflammation and apoptosis of cardiomyocytes through regulating miR-16 demethylation
Immunity, Inflammation and Disease ( IF 2.493 ) Pub Date : 2021-09-21 , DOI: 10.1002/iid3.498
Yan Wang 1 , Yi Zhang 1
Affiliation  

The long noncoding RNA, cardiac autophagy inhibitory factor (CAIF), and microRNA (miR)-16 are reported to be involved in lipopolysaccharide (LPS)-induced inflammatory responses and cell apoptosis in many diseases. Herein, we investigated the interaction between CAIF and miR-16 in sepsis-induced chronic heart failure (CHF).

中文翻译:

LncRNA CAIF通过调节miR-16去甲基化抑制LPS诱导的心肌细胞炎症和凋亡

据报道,长链非编码 RNA、心脏自噬抑制因子 (CAIF) 和微小 RNA (miR)-16 参与脂多糖 (LPS) 诱导的炎症反应和许多疾病的细胞凋亡。在此,我们研究了 CAIF 和 miR-16 在败血症诱导的慢性心力衰竭 (CHF) 中的相互作用。
更新日期:2021-11-13
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