A 33-year-old man had verapamil-sensitive ventricular tachycardia (VT) with a right bundle branch block (RBBB) and right axis deviation. Programmed stimulation from the para-Hisian region induced ventricular tachycardias (VT1 or VT2). VT1 was entrained during pacing from the para-Hisian region. A single para-Hisian stimulation antidromically captured the proximal portion of the left anterior fascicle (LAF), but the cycle length of VT2 remained unchanged. This observation indicated that the upper limb of the LAF was a bystander of the reentry circuit. We have clarified this mechanism with applying a single premature stimulation from the para-Hisian region.

一名 33 岁男性患有维拉帕米敏感性室性心动过速 (VT)，伴右束支传导阻滞 (RBBB) 和电轴右偏。来自 para-Hisian 区域的程序刺激引起室性心动过速（VT1 或 VT2）。VT1 在从 para-Hisian 区域起搏期间被夹带。单个 para-Hisian 刺激逆向捕获左前束 (LAF) 的近端部分，但 VT2 的周期长度保持不变。这一观察表明 LAF 的上肢是再入回路的旁观者。我们已经通过应用来自 para-Hisian 区域的单个过早刺激阐明了这种机制。