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Cover Image: Salmonella Typhimurium manipulates macrophage cholesterol homeostasis through the SseJ-mediated suppression of the host cholesterol transport protein ABCA1 (Cellular Microbiology 08/2021)
Cellular Microbiology ( IF 3.4 ) Pub Date : 2021-07-13 , DOI: 10.1111/cmi.13377
Adam R. Greene , Katherine A. Owen , James E. Casanova

Salmonella Typhimurium induces accumulation of cholesterol in WT macrophages (top right) but not in the absence of the host kinase FAK (bottom left) or the bacterial effector protein SseJ (bottom right). For further details, readers are referred to the article by Greene et al. on p. e13329 of this issue.
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中文翻译:

封面图片:鼠伤寒沙门氏菌通过 SseJ 介导的宿主胆固醇转运蛋白 ABCA1 的抑制来操纵巨噬细胞胆固醇稳态(细胞微生物学 08/2021)

鼠伤寒沙门氏菌诱导 WT 巨噬细胞中胆固醇的积累(右上),但在没有宿主激酶 FAK(左下)或细菌效应蛋白 SseJ(右下)的情况下不会。有关更多详细信息,读者可以参考 Greene 等人的文章。在页。这个问题的e13329。
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更新日期:2021-07-13
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