This study focused on the redox homeostasis in the lenses of 20- and 30-day-old chinchilla rabbit kits exposed to a combination of hypoxia and electromagnetic radiation at a nonthermal intensity during their prenatal development. Adult rabbit does (n = 12) were divided into equal-sized experimental and control groups. After mating, pregnant animals from the experimental group were exposed to hypoxia and electromagnetic radiation for 6 days, while the control animals were sham-irradiated but not exposed to hypoxia. After birth the newborn rabbits were also divided into the experimental and control groups. Kits were slaughtered on the 20th and 30th days after birth and the lenses were removed. The lipid peroxidation intensity was estimated by the measurement of malondialdehyde in the lens tissue specimens. It was found that the redox status in the lenses of experimental rabbit kits changed towards oxidation, probably due to failure in the antioxidant defense mechanism. Thus, it was proven that prenatally induced disturbance of redox homeostasis negatively affected the postnatal period as well. No significant differences were found between the extent of lipid peroxidation by the 20th and 30th days of the postnatal period.

这项研究的重点是 20 天和 30 天大的龙猫兔套件在其产前发育期间暴露于缺氧和电磁辐射的组合中的晶状体中的氧化还原稳态。成年兔 ( n= 12) 被分成相等大小的实验组和对照组。交配后，实验组的怀孕动物在缺氧和电磁辐射下暴露6天，而对照组动物进行假照射但不暴露在缺氧环境中。出生后的新生兔也被分为实验组和对照组。在出生后第 20 天和第 30 天屠宰套件并取下镜片。通过测量晶状体组织样本中的丙二醛来估计脂质过氧化强度。发现实验兔试剂盒镜片中的氧化还原状态转变为氧化，可能是由于抗氧化防御机制的失败。因此，证明出生前引起的氧化还原稳态紊乱也会对出生后时期产生负面影响。