The etiology of borderline personality pathology has consistently been framed as an interactional process between child vulnerability (i.e. emotional sensitivity and reactivity; Linehan, 1993) and invalidating parenting strategies, which evolves into increased emotion dysregulation and disinhibited behavior of the child and in turn activates more parental invalidation. Despite the strong theoretical base in support of these high-risk parent–child transactions, invalidating parenting behaviors have mostly been explored as a cause of child dysregulation and disinhibition, rather than as a result of child-driven effects. Also, most transactional research in this regard focused at differences between families, thereby not addressing potential changes within families across time. The current study therefore examines bidirectional between- and within-family effects of childhood borderline-related traits and maternal invalidation in the sensitive developmental phase of pre-adolescence (n = 574; 54.4% girls) along three assessment points. Cross-Lagged Panel Models and Random-Intercept Cross-Lagged Panel Models indicated detrimental parenting effects of invalidation on subsequent development in borderline-related traits of the child both between and within families, and additional child-driven effects for subsequent invalidating parenting strategies within families. Beyond these transactions between borderline-related traits and parenting, the current study also indicates significant differences in the direction of effects when exploring transactions between more common dimensions of child internalizing/externalizing symptomatology and parental invalidation, suggesting a more substantial parenting etiology in the developmental process of borderline traits throughout pre-adolescence. Future longitudinal research may explore to what extent the transactional nature of borderline personality traits during important developmental stages indeed holds unique aspects compared to more common manifestations of symptomatology at young age.

边缘人格病理的病因学一直被认为是儿童脆弱性（即情绪敏感性和反应性； Linehan，1993）与无效养育策略之间的相互作用过程，其演变成情绪失调和儿童行为异常，进而激活了更多的儿童。父母无效。尽管有强有力的理论基础支持这些高风险的亲子交易，但无效的养育行为大多被认为是造成儿童失调和禁忌的原因，而不是儿童驱动的结果。同样，在这方面，大多数交易研究都集中在家庭之间的差异上，因此没有解决家庭内部随着时间的推移可能发生的变化。ñ = 574; 54.4％的女孩）和三个评估点。交叉滞后的面板模型和随机拦截交叉滞后的面板模型表明，无效对家庭之间和家庭内部儿童边界相关特征的继发发展的有害育儿效应，以及随后由家庭导致的无效育儿策略的其他儿童驱动效应。除了边界相关性状和育儿之间的这些交易外，当前的研究还表明，在探索儿童内化/外化症状学与父母无效的更常见维度之间的交易时，在影响方向上存在显着差异，这表明在发展过程中更重要的育儿病因整个青春期前的边缘特征。