COVID-19 is a severe respiratory disorder caused by the SARS COV-2 virus that involves limited innate immunity. Numerous publications have suggested that plants/minerals used in the traditional system of Ayurveda, has revealed much about the biology of COVID-19. One theory is that combination of anti viral, anti inflammatory, agents activating immune cells, herbs and metals may be helpful for severe acute respiratory syndrome coronavirus infection. Anti-viral drugs used for COVID-19 are those which block RNA synthesis and virus invasion, and bind to receptor proteins on the surface of cells, cell cycle protein, and physiological and pathological processes inhibitor. Anti-inflammatory drugs used for COVID-19 are those which controls transcription of DNA, cytokine production, break down the basement membrane, regulate outer mitochondrial membrane permeability, controlling the host cell life, stimulates activated B-cell and T-cell proliferation, virus dissemination, a slowdown of cell metabolism or secretion of cytokines. Drugs which is having role in the innate immunity, inhibits ROS, enhances cell lifespan, activates macrophages, physiological effects on cells activates the Lung resident immune cells. The focus of this review is to elucidate the Ayurvedic pharmacological properties with their therapeutic targets.

中文翻译：

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通过传统的阿育吠陀药物系统确定控制COVID-19大流行的治疗靶点：系统综述

COVID-19是由SARS COV-2病毒引起的严重呼吸系统疾病，涉及有限的先天免疫。许多出版物建议在阿育吠陀传统体系中使用的植物/矿物质，已经揭示了许多有关COVID-19的生物学信息。一种理论是抗病毒，抗炎，激活免疫细胞，草药和金属的试剂的组合可能有助于严重的急性呼吸系统综合症冠状病毒感染。用于COVID-19的抗病毒药是那些阻断RNA合成和病毒入侵并与细胞表面的受体蛋白，细胞周期蛋白以及生理和病理过程抑制剂结合的药物。用于COVID-19的抗炎药是控制DNA转录，细胞因子产生，分解基底膜，调节线粒体外膜通透性，控制宿主细胞寿命，刺激活化的B细胞和T细胞增殖，病毒的药物。传播，细胞代谢减慢或细胞因子分泌。在先天免疫中起作用的药物，可抑制ROS，延长细胞寿命，激活巨噬细胞，对细胞的生理作用可激活肺部驻留免疫细胞。这次审查的重点是阐明阿育吠陀的药理特性及其治疗目标。