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Bidirectional associations between alcohol use and intimate partner violence and sexual assault victimization among college women
Addictive Behaviors ( IF 4.4 ) Pub Date : 2021-01-16 , DOI: 10.1016/j.addbeh.2021.106833
Christina M Dardis 1 , Sarah E Ullman 2 , Lindsey M Rodriguez 3 , Emily A Waterman 4 , Emily R Dworkin 5 , Katie M Edwards 6
Affiliation  

Whereas some prior studies have explored whether alcohol increases the risk for victimization and/or whether distress resulting from victimization increases the risk for alcohol use, few studies have simultaneously tested these bidirectional hypotheses among a high-risk sample (i.e., undergraduate women), while including both sexual assault (SA) and intimate partner violence (IPV) victimization, and exploring potential moderating effects of PTSD symptoms on these paths. Among 631 college women, the present study tested these bidirectional associations using cross-lagged panel models across two measurement periods (i.e., Time 1 [T1] and Time 2 [T2], six months later). Results suggested that T1 alcohol use increased risk for T2 SA (but not T2 IPV victimization), and PTSD symptoms moderated this association; at lower levels of PTSD symptoms, there were no significant associations between alcohol use and subsequent SA victimization, whereas at higher levels of PTSD symptoms, alcohol use predicted subsequent SA victimization. By contrast, the opposite directional hypothesis was not supported; neither T1 lifetime SA nor IPV were associated with T2 drinking, regardless of the level of their PTSD symptoms. Prevention and intervention efforts should simultaneously address risk factors for alcohol use and victimization using trauma-informed practices.



中文翻译:

大学女性酒精使用与亲密伴侣暴力和性侵犯受害之间的双向关联

尽管一些先前的研究已经探讨了酒精是否会增加受害的风险和/或受害导致的痛苦是否会增加饮酒的风险,但很少有研究同时在高风险样本(即本科女性)中检验这些双向假设,而包括性侵犯 (SA) 和亲密伴侣暴力 (IPV) 受害,并探索 PTSD 症状对这些途径的潜在调节作用。在 631 名大学女性中,本研究使用跨两个测量期(即时间 1 [T1] 和时间 2 [T2],六个月后)的交叉滞后面板模型测试了这些双向关联。结果表明,T1 酒精使用增加了 T2 SA 的风险(但不是 T2 IPV 受害),并且 PTSD 症状缓和了这种关联;在较低水平的 PTSD 症状,酒精使用与随后的 SA 受害之间没有显着关联,而在更高水平的 PTSD 症状中,酒精使用预测随后的 SA 受害。相比之下,不支持相反方向的假设;无论他们的 PTSD 症状水平如何,T1 终生 SA 和 IPV 都与 T2 饮酒无关。预防和干预工作应同时解决使用创伤知情实践的酒精使用和受害风险因素。无论他们的 PTSD 症状水平如何。预防和干预工作应同时解决使用创伤知情实践的酒精使用和受害风险因素。无论他们的 PTSD 症状水平如何。预防和干预工作应同时解决使用创伤知情实践的酒精使用和受害风险因素。

更新日期:2021-01-28
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