In developing an effective clinical tool against COVID-19, we need to consider why SARS-CoV-2 infections develop along remarkably different trajectories: from completely asymptomatic to a severe course of disease. In this paper we hypothesize that the progressive exhaustion and loss of lymphocytes associated with severe stages of COVID-19 result from an intracellular energy deficit in an organism which has already been depleted by preexisting chronic diseases, acute psychological stress and the aging process. A bioenergetics view of COVID-19 immunopathology opens a new biophysical opportunity to enhance impaired immune function via proposed pathways of photomagnetic catalysis of ATP synthesis, regenerative photobiomodulation and the ultrasonic acceleration of cell restructuring. Moreover, we suggest that a coherent application of multiple biophysical radiances (coMra) may synergistically enhance energy-matter-information kinetics of basal self-regeneration of cells and thus improve immune function and accelerate recovery.

在开发针对COVID-19的有效临床工具时，我们需要考虑为什么SARS-CoV-2感染会沿着截然不同的轨迹发展：从完全无症状到严重的病程。在本文中，我们假设与COVID-19严重阶段相关的淋巴细胞的逐渐耗尽和丢失是由于生物体内的细胞内能量不足引起的，而该能量已经被预先存在的慢性疾病，急性心理压力和衰老过程所消耗。COVID-19免疫病理学的生物能学观点为通过拟议的ATP合成，再生光生物调节和超声加速细胞重组的光磁催化途径增强免疫功能受损提供了新的生物物理机会。此外，