Purpose of review 

The aim of this study was to highlight the recent advancements and future directions for potential use of a low carbohydrate ketogenic dietary approach to treat binge eating and ultraprocessed food addiction. Herein, we explore proposed mechanisms of why a diet low in refined carbohydrates, processed sugar and higher fat content may be helpful in alleviating symptoms.

Recent findings 

Emerging evidence suggests there may be a metabolic role in development of maladaptive eating. These findings broaden our understanding of eating psychopathology causes. Ultraprocessed, refined or high glycemic index carbohydrates are a possible trigger mediating neurochemical responses similar to addiction. The carbohydrate-insulin model of obesity supports observations of these foods triggering abnormal blood sugar and insulin spikes subsequently leading to changes in metabolic and neurobiological signaling. This results in overeating symptoms and hunger exacerbation, which differs from observed effects of healthy fat consumption and lack of similar insulin spikes. As supported in recent case series, significantly reducing or abstaining from these addictive-like ultraprocessed foods and highly refined carbohydrates could be considered a treatment approach.

Summary 

The current review highlights recent and pertinent evidence with respect to theoretical and practical application of low carbohydrate ketogenic therapeutic approaches for ultraprocessed food addiction and binge eating symptoms.

http://links.lww.com/COE/A18

中文翻译：

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低碳水化合物生酮疗法可作为暴食和超加工食物成瘾的代谢疗法

审查目的 

这项研究的目的是强调低碳水化合物生酮饮食疗法的最新进展和未来方向，可能用于治疗暴饮暴食和超加工食物成瘾。本文中，我们探讨了为什么精制碳水化合物，加工糖和脂肪含量较低的饮食可能有助于缓解症状的机制。

最近的发现 

新兴证据表明，不良适应饮食的发展可能具有代谢作用。这些发现拓宽了我们对饮食心理病理原因的理解。超加工，精制或高血糖指数的碳水化合物可能是介导类似于成瘾的神经化学反应的触发因素。肥胖的碳水化合物-胰岛素模型支持对这些食物引发异常血糖和胰岛素峰值的观察，随后导致代谢和神经生物学信号的改变。这导致暴饮暴食的症状和饥饿加剧，这与观察到的健康脂肪消耗和缺乏类似胰岛素峰值的影响不同。正如最近的一系列病例所支持的那样，可以将这些成瘾性的超加工食品和高度精制的碳水化合物大量减少或戒除可以视为一种治疗方法。

概要 

本篇评论着重介绍了低碳水化合物生酮治疗方法在超加工食品成瘾和暴饮暴食症状方面的理论和实际应用方面的最新和相关证据。

http://links.lww.com/COE/A18