In this issue of Immunity, Xu et al. reveal that dermal dendritic cells produce interleukin-31, which acts on neurons to promote wound itch. Their findings link itch associated with deeper wounds—wounds that extend beyond the epithelium—to the cells and cytokines that mediate wound healing.

中文翻译：

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在发痒的伤口表面刮擦。

在本期《免疫》中，Xu等人。揭示真皮树突状细胞产生白介素31，其作用于神经元以促进伤口瘙痒。他们的发现将与深层伤口相关的痒（伤口延伸到上皮之外）与介导伤口愈合的细胞和细胞因子联系在一起。