Our cover features an H&E staining of lung sections from diesel exhaust particles‐ intranasally administered WT mice. The image is taken from Li et al. (pp. 1142–1153), where the authors present the pulmonary inflammation induced by diesel exhaust particles administration is attenuated in the absence of IRF3 and IRF7 due to increased autophagy activity, providing novel insights into the mechanism of autophagy formation and potential therapeutic approaches to counteract the adverse effects of diesel exhaust particles and other ambient particular matters.

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封面故事：Eur。J.免疫。8'20

我们的封面的特点是通过柴油机尾气颗粒鼻内给药的野生型小鼠的肺切片进行H＆E染色。该图像取自Li等人。（pp。1142–1153），作者提出由于自噬活性增强，在不存在IRF3和IRF7的情况下，柴油机排气微粒引起的肺部炎症减弱，为自噬形成的机理和潜在的治疗方法提供了新见解。抵消柴油机排气颗粒和其他环境特殊问题的不利影响。