Metabolic support for regulatory T (Treg) cells in noninflamed tumors is not well understood. In this issue of Immunity, Kumagai et. al. show how oncogene-driven fatty-acid synthesis favors Treg cells over effector T cells and how this imbalance can be overcome.

中文翻译：

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致癌基因喂养Treg细胞而无需在表中调用CD8。

对于非炎性肿瘤中调节性T（Treg）细胞的代谢支持尚不清楚。在本期《免疫》中，熊谷等人 等 展示了癌基因驱动的脂肪酸合成如何比效应T细胞更偏爱Treg细胞，以及如何克服这种不平衡。