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Keratinocyte-Polyamines and Dendritic Cells: A Bad Duet for Psoriasis.
Immunity ( IF 32.4 ) Pub Date : 2020-07-14 , DOI: 10.1016/j.immuni.2020.06.015 Mohna Bandyopadhyay 1 , Adriana T Larregina 2
中文翻译:
角质形成细胞多胺和树突状细胞:银屑病的二重奏。
更新日期:2020-07-14
Immunity ( IF 32.4 ) Pub Date : 2020-07-14 , DOI: 10.1016/j.immuni.2020.06.015 Mohna Bandyopadhyay 1 , Adriana T Larregina 2
Affiliation
The role of keratinocyte metabolism in psoriasis is not fully elucidated. In this issue of Immunity, Lou et al. describe that interleukin-17 (IL-17) re-programs the urea cycle in keratinocytes increasing polyamines that stabilize RNA-Ag-complexes that upon cellular turnover activate dendritic cells, which amplify psoriasis inflammation.
中文翻译:
角质形成细胞多胺和树突状细胞:银屑病的二重奏。
尚未完全阐明角质形成细胞代谢在牛皮癣中的作用。在《免疫》杂志中,Lou等人 文献描述了白介素17(IL-17)重新编程了角质形成细胞中的尿素循环,从而增加了稳定RNA-Ag复合物的多胺,该复合物在细胞更新时激活树突状细胞,从而放大了牛皮癣的炎症。