As many as 80% of patients with TAR die on the spot while out of those reaching a hospital, 30% would die within 24 hours. Thus, it is essential to better understand and prevent this injury. The exact mechanics of TAR are unknown. Although most researchers approve it as a common-sense deceleration injury, the exact detailed mechanism of TRA still remains unidentified. In this work, a deceleration mechanism of TAR was carried out using finite element analysis (FEA). The FE analysis aimed to predict internal kinematics of the aorta and assist to comprehend the mechanism of aorta injury. The model contains the heart, lungs, thoracic aorta vessel, and rib cage. High-resolution computerized tomography (HR CT scan) was used to provide pictures that were reconstructed by MIMICS software. ANSYS FE simulation was carried out to investigate the behavior of the aorta in the thoracic interior after deceleration occurred during a car crash. The finite element analysis indicated that maximum stress and strain applied to the aorta were from 5.4819e5 to 2.614e6 Pa and 0.21048 to 0.62676, respectively, in the Y-direction when the initial velocity increased from 10 to 25 m/s. Furthermore, in the X-direction when the velocity changed from 15 to 25 m/s, the stress and strain values increased from 5.17771e5 to 2.3128e6 and from 0.22445 to 0.618, respectively.

中文翻译：

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创伤性主动脉破裂机制的有限元分析。

多达80％的TAR患者当场死亡，而在送往医院的患者中，有30％会在24小时内死亡。因此，必须更好地理解和防止这种伤害。TAR的确切机理尚不清楚。尽管大多数研究人员都将其视为常识性减速伤害，但TRA的确切机制仍不清楚。在这项工作中，使用有限元分析（FEA）进行了TAR的减速机制。有限元分析旨在预测主动脉的内部运动学，并有助于了解主动脉损伤的机制。该模型包含心脏，肺部，胸主动脉血管和肋骨笼。高分辨率计算机断层扫描（HR CT扫描）用于提供由MIMICS软件重建的图片。进行了ANSYS FE模拟，以研究车祸后减速后胸腔内主动脉的行为。有限元分析表明，在主动脉中，施加于主动脉的最大应力和应变分别为5.4819e5至2.614e6 Pa和0.21048至0.62676。初始速度从10 m / s增加到25 m / s时的Y方向。此外，当速度从15变为25 m / s时，在X方向上，应力和应变值分别从5.17771e5增大到2.3128e6，从0.22445增大到0.618。