Myocardial infarction is one of the major causes of death worldwide. Many heart cells die during myocardial infarction through various processes such as necrosis, apoptosis, necroptosis, autophagy-related cell death, pyroptosis and ferroptosis. These dead cells in infarcted hearts expose the so-called ‘eat-me’ signals, such as phosphatidylserine, on their surfaces, enhancing their removal by professional and non-professional phagocytes. Clearance of dead cells by phagocytes in the diseased hearts plays a crucial role in the pathology of myocardial infarction by inhibiting the inflammatory responses caused by the leakage of contents from dead cells. This review focuses on the rapidly growing understanding of the molecular mechanisms of dead cell phagocytosis, termed efferocytosis, during myocardial infarction, which contributes to the pathophysiology of myocardial infarction.

中文翻译：

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心肌梗死时的红细胞增多症。

心肌梗塞是全世界主要的死亡原因之一。在心肌梗塞期间，许多心脏细胞通过各种过程死亡，例如坏死，凋亡，坏死病，自噬相关的细胞死亡，发烧和铁生病。梗塞心脏中的这些死细胞在其表面暴露出所谓的“ eat-me”信号，例如磷脂酰丝氨酸，从而增强了它们被专业和非专业吞噬细胞清除的能力。通过抑制由死细胞内容物泄漏引起的炎症反应，患病心脏中吞噬细胞清除死细胞在心肌梗死的病理中起着至关重要的作用。这篇综述着重于对心肌梗死期间死亡细胞吞噬作用（称为胞吞作用）的分子机制的迅速增长的了解，