To evaluate the rapid repair potential of adipose‐derived stem cells (ADSCs) co‐overexpressing VEGF and GDNF on bilateral cavernous nerve injury (BCNI) in rat models. Progressive fibrosis of the penis that occurs shortly after BCNI is a key cause of clinical treatment difficulty of erectile dysfunction (ED). Traditional medications are ineffective for ED caused by BCNI. ADSCs have shown therapeutic effects in animal models, but disappointing in clinical treatment suggests that we should explore optimal treatment of it.

中文翻译：

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脂肪干细胞中VEGF和GDNF的共过表达优化了神经源性勃起功能障碍模型的治疗效果

评估脂肪干细胞 (ADSCs) 共过表达 VEGF 和 GDNF 对大鼠双侧海绵状神经损伤 (BCNI) 的快速修复潜力。BCNI 后不久发生的阴茎进行性纤维化是勃起功能障碍 (ED) 临床治疗困难的​​关键原因。传统药物对 BCNI 引起的 ED 无效。ADSCs 已在动物模型中显示出治疗效果，但临床治疗令人失望表明我们应该探索其最佳治疗方法。