Acute heat shock has previously been shown to improve subsequent low O₂ (hypoxia) tolerance in an intertidal fish species, a process known as cross-tolerance, but it is not known whether this is a widespread phenomenon. This study examined whether a rock pool specialist, the triplefin fish Bellapiscis medius, exhibits heat shock induced cross-tolerance to hypoxia, i.e., longer time to loss of equilibrium (LOE) and lower critical O₂ saturation (S_crit) after recovering from an acute heat challenge. Non-heat shock controls had a median time to loss of equilibrium (LOE₅₀) of 54.4 min under severe hypoxia (7% of air saturation) and a S_crit of 15.8% air saturation. Contrary to expectations, however, treatments that received an 8 or 10°C heat shock showed a significantly shorter LOE₅₀ in hypoxia (+8°C = 41.5 min; +10°C = 28.7 min) and no significant change in S_crit (+8°C = 17.0% air saturation; +10°C = 18.3% of air saturation). Thus, there was no evidence of heat shock induced cross-tolerance to hypoxia in B. medius because exposure to acute heat shock impaired hypoxia tolerance.

先前已经显示，急性热激可以改善潮间鱼类的低O ₂（低氧）耐受性，这一过程称为交叉耐受，但尚不清楚这是否是普遍现象。这项研究检查了岩石池专家三鳍鱼Bellapiscis medius是否表现出热激诱导的对缺氧的交叉耐受性，即从水体中恢复后失去平衡的时间更长（LOE）和临界O ₂饱和度更低（S _crit）。急性热挑战。非热休克控制的中位时间来平衡损耗（LOE ₅₀的严重缺氧下54.4分钟）（空气饱和度的7％）和A S_暴15.8％的空气饱和度。然而，与预期相反，接受8或10°C热休克的治疗显示缺氧时LOE ₅₀明显缩短（+ 8°C = 41.5分钟； + 10°C = 28.7分钟），S_临界值无明显变化（ + 8°C = 17.0％的空气饱和度； + 10°C = 18.3％的空气饱和度）。因此，没有证据表明热休克对B的缺氧有交叉耐受性。medius，因为暴露于急性热休克削弱了低氧耐受性。