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Salicylic acid antagonizes selenium phytotoxicity in rice: selenium homeostasis, oxidative stress metabolism and methylglyoxal detoxification.
Journal of Hazardous Materials ( IF 13.6 ) Pub Date : 2020-03-20 , DOI: 10.1016/j.jhazmat.2020.122572
Mohammad Golam Mostofa 1 , Md Mezanur Rahman 2 , Md Nurealam Siddiqui 1 , Masayuki Fujita 3 , Lam-Son Phan Tran 4
Affiliation  

We investigated the mechanistic consequences of selenium (Se)-toxicity, and its possible mitigation using salicylic acid (SA) in rice. In comparison with control, sodium selenate-exposed 'Se1' (0.5 mM) and 'Se2' (1.0 mM) plants showed accumulation of Se by 190.63 and 288.00 % in roots, 2359.42 and 2054.35 % in leaf sheaths, and 7869.91 and 9063.72 % in leaves, respectively, resulting in severe toxicity symptoms, such as growth inhibition, chlorosis, burning of leaves, and oxidative stress. In contrast, SA addition to Se-stressed plants significantly alleviated the Se-toxicity symptoms, and radically improved shoot height (28.88 %), dry biomass (34.00 %), total chlorophyll (37.51 %), soluble sugar (17.31 %) and leaf water contents (22.31 %) in 'SA + Se2' plants over 'Se2' plants. Notably, SA maintained Se-homeostasis, and decreased 'Se2'-induced oxidative stress by enhancing ascorbate level (67.75 %) and the activities of antioxidant enzymes like superoxide dismutase (20.99 %), catalase (40.97 %), glutathione peroxidase (12.26 %), and glutathione reductase (32.58 %) relative to that in 'Se2' plants. Additionally, SA protected rice plants from the deleterious effects of methylglyoxal by stimulating the activities of glyoxalase enzymes. Furthermore, SA upregulated several genes associated with reactive oxygen species (e.g. OsCuZnSOD1, OsCATB, OsGPX1 and OsAPX2) and methylglyoxal (e.g. OsGLYI-1) detoxifications. These findings unravel a decisive role of SA in alleviating Se-phytotoxicity in rice.

中文翻译:

水杨酸拮抗水稻中硒的植物毒性:硒体内稳态,氧化应激代谢和甲基乙二醛解毒。

我们调查了硒(Se)毒性的机械性后果,以及在水稻​​中使用水杨酸(SA)缓解硒的可能性。与对照相比,暴露于硒酸钠的'Se1'(0.5 mM)和'Se2'(1.0 mM)植物的根系中硒的累积量分别为190.63和288.00%,叶鞘中的2359.42和2054.35%以及7869.91和9063.72%分别导致叶片中严重的毒性症状,如生长抑制,萎黄,叶片燃烧和氧化应激。相比之下,除硒胁迫植物外,SA还显着减轻了Se的毒性症状,从根本上改善了苗高(28.88%),干燥生物量(34.00%),总叶绿素(37.51%),可溶性糖(17.31%)和叶片与“ Se2”植物相比,“ SA + Se2”植物中的水分含量(22.31%)。值得注意的是,SA维持了Se稳态,并通过提高抗坏血酸水平(67.75%)和抗氧化剂酶(如超氧化物歧化酶(20.99%),过氧化氢酶(40.97%),谷胱甘肽过氧化物酶(12.26%)和谷胱甘肽还原酶(32.58%)的活性来降低'Se2'诱导的氧化应激。 )(相对于“ Se2”植物中的)。另外,SA通过刺激乙二醛酶的活性来保护稻株免受甲基乙二醛的有害作用。此外,SA上调了与活性氧种类(例如OsCuZnSOD1,OsCATB,OsGPX1和OsAPX2)和甲基乙二醛(例如OsGLYI-1)相关的几种基因。这些发现揭示了SA在减轻水稻中Se-植物毒性中的决定性作用。与'Se2'植物相比,抗氧化酶的活性为75%)和超氧化物歧化酶(20.99%),过氧化氢酶(40.97%),谷胱甘肽过氧化物酶(12.26%)和谷胱甘肽还原酶(32.58%)的活性。另外,SA通过刺激乙二醛酶的活性来保护稻株免受甲基乙二醛的有害作用。此外,SA上调了与活性氧(例如OsCuZnSOD1,OsCATB,OsGPX1和OsAPX2)和甲基乙二醛(例如OsGLYI-1)解毒相关的几个基因。这些发现揭示了SA在减轻水稻中Se-植物毒性中的决定性作用。与'Se2'植物相比,抗氧化酶的活性为75%)和超氧化物歧化酶(20.99%),过氧化氢酶(40.97%),谷胱甘肽过氧化物酶(12.26%)和谷胱甘肽还原酶(32.58%)的活性。另外,SA通过刺激乙二醛酶的活性来保护稻株免受甲基乙二醛的有害作用。此外,SA上调了与活性氧(例如OsCuZnSOD1,OsCATB,OsGPX1和OsAPX2)和甲基乙二醛(例如OsGLYI-1)解毒相关的几个基因。这些发现揭示了SA在减轻水稻中Se-植物毒性中的决定性作用。SA通过刺激乙二醛酶的活性来保护水稻免受甲基乙二醛的有害影响。此外,SA上调了与活性氧(例如OsCuZnSOD1,OsCATB,OsGPX1和OsAPX2)和甲基乙二醛(例如OsGLYI-1)解毒相关的几个基因。这些发现揭示了SA在减轻水稻中Se-植物毒性中的决定性作用。SA通过刺激乙二醛酶的活性来保护水稻免受甲基乙二醛的有害影响。此外,SA上调了与活性氧(例如OsCuZnSOD1,OsCATB,OsGPX1和OsAPX2)和甲基乙二醛(例如OsGLYI-1)解毒相关的几个基因。这些发现揭示了SA在减轻水稻中Se-植物毒性中的决定性作用。
更新日期:2020-03-21
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