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Epicardial adipose tissue is a predictor of ascending aortic dilatation in hypertensive patients, but not paracardial adipose tissue
BMC Cardiovascular Disorders ( IF 2.1 ) Pub Date : 2020-03-19 , DOI: 10.1186/s12872-020-01431-2 Onur Argan , Eyup Avci , Tarik Yildirim , Ozgen Safak
BMC Cardiovascular Disorders ( IF 2.1 ) Pub Date : 2020-03-19 , DOI: 10.1186/s12872-020-01431-2 Onur Argan , Eyup Avci , Tarik Yildirim , Ozgen Safak
Ascending aortic aneurysms are one of the major causes of mortality. In recent years, there is a growing interest of epicardial adipose tissue (EAT) and related diseases. The aim of this study was to investigate the relationship of EAT, and PAT with ascending aortic dilatation (AAD). We included 934 patients with hypertension in this study. The patients were evaluated by a complete transthoracic echocardiographic examination, including measurements of EAT, PAT, and aortic dimensions. Aortic size index (ASI) was used for diagnosing AAD. The patients were divided into two groups: dilated ascending aorta diameter (ASI ≥ 21 mm / m2, n = 102) or normal aortic diameter (ASI < 21 mm / m2, n = 832) according to the ASI. Characteristics of these patients were compared before and after propensity score matching analysis. Patients with AAD were older (72.3 ± 11.6 vs. 61.7 ± 12.7 years, p < 0.001), had more female gender (66% vs. 54%,p = 0.021) than patients with normal ascending aorta (AA). After propensity score matching analysis (77 vs. 77), EAT [OR:1.461, %95CI (1.082–1.974), p = 0.013] was independently associated with AAD in multivariate logistic regression analysis. In ROC curve analysis, EAT > 0.45 cm had 51.9% sensitivity and 62.3% specificity [AUC = 0.617, P = 0.012, 95% CI (0.529–0.707)]. Based on our findings, increased EAT may be suggested as an independent risk factor for AAD due to local or systemic effects in hypertensive patients.
中文翻译:
心外膜脂肪组织是高血压患者升主动脉扩张的预测因子,但心包脂肪组织则不
升主动脉瘤是死亡的主要原因之一。近年来,心外膜脂肪组织(EAT)和相关疾病的兴趣日益浓厚。这项研究的目的是研究EAT和PAT与升主动脉扩张(AAD)的关系。在这项研究中,我们纳入了934例高血压患者。通过完整的经胸超声心动图检查对患者进行评估,包括对EAT,PAT和主动脉尺寸的测量。主动脉大小指数(ASI)用于诊断AAD。根据ASI将患者分为两组:扩张的升主动脉直径(ASI≥21 mm / m2,n = 102)或正常的主动脉直径(ASI <21 mm / m2,n = 832)。比较倾向评分匹配分析前后这些患者的特征。AAD患者年龄较大(72。3±11.6岁vs. 61.7±12.7岁,p <0.001)比正常升主动脉(AA)患者的女性性别多(66%vs. 54%,p = 0.021)。经过倾向得分匹配分析(77比77),在多因素logistic回归分析中,EAT [OR:1.461,%95CI(1.082-1.974),p = 0.013]与AAD独立相关。在ROC曲线分析中,EAT> 0.45 cm具有51.9%的敏感性和62.3%的特异性[AUC = 0.617,P = 0.012,95%CI(0.529-0.707)]。根据我们的发现,由于高血压患者的局部或全身作用,饮食增加可能被认为是AAD的独立危险因素。013]在多元Logistic回归分析中与AAD独立相关。在ROC曲线分析中,EAT> 0.45 cm具有51.9%的敏感性和62.3%的特异性[AUC = 0.617,P = 0.012,95%CI(0.529-0.707)]。根据我们的发现,由于高血压患者的局部或全身作用,饮食增加可能被认为是AAD的独立危险因素。013]在多元Logistic回归分析中与AAD独立相关。在ROC曲线分析中,EAT> 0.45 cm具有51.9%的敏感性和62.3%的特异性[AUC = 0.617,P = 0.012,95%CI(0.529-0.707)]。根据我们的发现,由于高血压患者的局部或全身作用,饮食增加可能被认为是AAD的独立危险因素。
更新日期:2020-04-22
中文翻译:
心外膜脂肪组织是高血压患者升主动脉扩张的预测因子,但心包脂肪组织则不
升主动脉瘤是死亡的主要原因之一。近年来,心外膜脂肪组织(EAT)和相关疾病的兴趣日益浓厚。这项研究的目的是研究EAT和PAT与升主动脉扩张(AAD)的关系。在这项研究中,我们纳入了934例高血压患者。通过完整的经胸超声心动图检查对患者进行评估,包括对EAT,PAT和主动脉尺寸的测量。主动脉大小指数(ASI)用于诊断AAD。根据ASI将患者分为两组:扩张的升主动脉直径(ASI≥21 mm / m2,n = 102)或正常的主动脉直径(ASI <21 mm / m2,n = 832)。比较倾向评分匹配分析前后这些患者的特征。AAD患者年龄较大(72。3±11.6岁vs. 61.7±12.7岁,p <0.001)比正常升主动脉(AA)患者的女性性别多(66%vs. 54%,p = 0.021)。经过倾向得分匹配分析(77比77),在多因素logistic回归分析中,EAT [OR:1.461,%95CI(1.082-1.974),p = 0.013]与AAD独立相关。在ROC曲线分析中,EAT> 0.45 cm具有51.9%的敏感性和62.3%的特异性[AUC = 0.617,P = 0.012,95%CI(0.529-0.707)]。根据我们的发现,由于高血压患者的局部或全身作用,饮食增加可能被认为是AAD的独立危险因素。013]在多元Logistic回归分析中与AAD独立相关。在ROC曲线分析中,EAT> 0.45 cm具有51.9%的敏感性和62.3%的特异性[AUC = 0.617,P = 0.012,95%CI(0.529-0.707)]。根据我们的发现,由于高血压患者的局部或全身作用,饮食增加可能被认为是AAD的独立危险因素。013]在多元Logistic回归分析中与AAD独立相关。在ROC曲线分析中,EAT> 0.45 cm具有51.9%的敏感性和62.3%的特异性[AUC = 0.617,P = 0.012,95%CI(0.529-0.707)]。根据我们的发现,由于高血压患者的局部或全身作用,饮食增加可能被认为是AAD的独立危险因素。