Current measures of anthracycline-induced cardiotoxicity, defined by decreases in left ventricular ejection fraction (LVEF), may become apparent only at a late stage when the myocardium has been significantly damaged, exceeding its ability to compensate ([1][1]). Our group has recently shown in a

中文翻译：

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T2 定位确定老年癌症患者的早期蒽环类药物诱导的心脏毒性。

由左心室射血分数 (LVEF) 降低定义的蒽环类药物引起的心脏毒性的当前措施可能仅在心肌已显着受损的晚期阶段变得明显，超过其代偿能力（[1][1]）。我们小组最近在一个