Inflammation has developed in the course of evolution as a process to defend the body against invading microbes and to respond to injuries. Several mechanisms of interaction between endothelial cells and leukocytes have evolved to render inflammation an effective, tightly controlled, and self-limited process. Imperfect executions of this "game plan" lead to pathological abnormalities resulting in diseases. The meeting on Molecular Mechanisms of Inflammation held at Schloss Elmau, Germany in October 2002 has featured activation of endothelial cells, adhesion and migration of leukocytes, as well as receptor pathways for activation and deactivation of leukocytes and, concomitantly, of the inflammatory response. Thus, a review on some of the presented data casts interesting spotlights on different steps of the inflammatory cascade.

中文翻译：

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会议报告：炎症的分子机制：白细胞如何出现，看见和抓住。

炎症是在进化过程中发展起来的，它是保护人体免受微生物侵袭并对伤害做出反应的过程。内皮细胞和白细胞之间相互作用的几种机制已经发展为使炎症成为有效，严格控制和自我限制的过程。该“游戏计划”执行不完善会导致导致疾病的病理异常。2002年10月在德国Schloss Elmau举行的炎症分子机制会议的主题是内皮细胞活化，白细胞粘附和迁移，以及白细胞活化和失活的受体途径以及随之而来的炎症反应。因此，对一些现有数据的评论将有趣的焦点投向了炎症级联反应的不同步骤。