we follow the publication regarding associations between FCGR polymorphisms and immune thrombocytopenia (ITP) with a great interest [1 - 2]. Li et al. firstly noted that "FCGR2A H131R polymorphism may serve as a potential genetic biomarker of childhood-onset ITP, while FCGR3A F158V polymorphism may serve as a potential genetic biomarker of both childhood-onset and adult-onset ITP [1]." As commented by Gholami et al., the study by Li et al. is a useful study on the effect of FCGR polymorphisms on ITP. Here, we would like to share additional ideas on this issue.

中文翻译：

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FCGR多态性与免疫性血小板减少症。

我们非常关注关于FCGR多态性与免疫性血小板减少症（ITP）之间的关联的出版物[1-2]。Li等。首先指出，“ FCGR2A H131R多态性可能是儿童期ITP的潜在遗传生物标志物，而FCGR3A F158V多态性可能是儿童期ITP和成年性ITP的潜在遗传生物标志物[1]。” 正如Gholami等人评论的那样，Li等人的研究表明。是一项关于FCGR多态性对ITP影响的有用研究。在这里，我们想就此问题分享其他想法。