Post‐occlusive reactive hyperemia (PORH) following arterial occlusion is widely used to assess cutaneous microvascular function, though the underlying mechanisms remain to be fully elucidated. We evaluated the hypothesis that Ca²⁺‐activated, ATP‐sensitive, and voltage‐gated K⁺ channels (K_Ca, K_ATP, and K_V channels, respectively) contribute to PORH while nitric oxide synthase (NOS) and cyclooxygenase (COX) do not.

中文翻译：

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四乙铵，格列本脲和4-氨基吡啶可调节无毛人皮肤中的闭塞后反应性充血，而没有NOS和COX的作用。

动脉闭塞后的闭塞后反应性充血（PORH）被广泛用于评估皮肤微血管功能，尽管其潜在机制尚待充分阐明。我们评估了以下假设：Ca ²⁺激活，ATP敏感和电压门控的K ⁺通道（分别为K _{C a}，K _ATP和K _V通道）有助于PORH，而一氧化氮合酶（NOS）和环氧合酶COX）不要。