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O2⋅− and H2O2-Mediated Disruption of Fe Metabolism Causes the Differential Susceptibility of NSCLC and GBM Cancer Cells to Pharmacological Ascorbate
Cancer Cell ( IF 50.3 ) Pub Date : 2017-08-14 00:00:00 , DOI: 10.1016/j.ccell.2017.07.008
Joshua D. Schoenfeld , Zita A. Sibenaller , Kranti A. Mapuskar , Brett A. Wagner , Kimberly L. Cramer-Morales , Muhammad Furqan , Sonia Sandhu , Thomas L. Carlisle , Mark C. Smith , Taher Abu Hejleh , Daniel J. Berg , Jun Zhang , John Keech , Kalpaj R. Parekh , Sudershan Bhatia , Varun Monga , Kellie L. Bodeker , Logan Ahmann , Sandy Vollstedt , Heather Brown , Erin P. Shanahan Kauffman , Mary E. Schall , Ray J. Hohl , Gerald H. Clamon , Jeremy D. Greenlee , Matthew A. Howard , Michael K. Schultz , Brian J. Smith , Dennis P. Riley , Frederick E. Domann , Joseph J. Cullen , Garry R. Buettner , John M. Buatti , Douglas R. Spitz , Bryan G. Allen

(Cancer Cell 31, 487–500; April 10, 2017)

中文翻译:


O2⋅-和H2O2介导的铁代谢破坏导致NSCLC和GBM癌细胞对药理抗坏血酸的敏感性不同

(癌细胞31,487-500; 2017年4月10日)

更新日期:2017-08-14
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